Cognitive & Nootropic
Preclinical
Research chemical

PE-22-28 (Spadin Analog)

Also known as PE-22-28 · Spadin analog · TREK-1 inhibitor peptide

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Reconstitution & dosing guide

10 mg vial — research phases, recon steps, and a live unit calculator.

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Available sizes & price

Catalog only

10mg x 10 vials

PE-22-28 · Not in current stock

$25 / vial

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Prices per vial · Research use only · Not medical advice.

Synthetic heptapeptide optimized from the natural sortilin-derived peptide spadin. Potent selective inhibitor of the TREK-1 (KCNK2) two-pore-domain potassium channel with preclinical antidepressant and neuroplasticity signals.

How to picture it

Neurons have background "leak" channels that let potassium slip out, which makes the cell more settled and harder to fire. TREK-1 is one of those calming leak channels. PE-22-28 is a potent blocker of TREK-1 — picture partially closing a pressure-relief valve so certain circuits (including mood-related ones in research) can regain dynamic range instead of staying stuck too quiet. Adjusting a neuron's idle valve, not flooding the brain with a broad stimulant.

Research focus & benefits

  • TREK-1 potassium-channel research
  • Mood and resilience circuit interest
  • Selective neuronal excitability studies

Benefits describe research interest and pathway rationale — not guaranteed outcomes or medical advice. Research use only.

Overview

PE-22-28 is a shortened, optimized analog of spadin (the PE 12–28 fragment released during sortilin maturation). Developed by French labs studying TREK-1 as an antidepressant target, it shows sub-nanomolar channel block and rapid behavioral effects in rodents. Human clinical data are essentially absent; it remains an early-stage research tool popular in nootropic catalogs.

Mechanism

Selectively blocks TREK-1 (KCNK2) with reported IC50 near 0.12 nM — roughly 300-fold more potent than parent spadin. TREK-1 blockade increases excitability of dorsal-raphe serotonergic neurons, elevates BDNF, phosphorylates CREB, and induces hippocampal neurogenesis within ~4 days in mouse models. Duration of antidepressant-like behavior reported up to ~23 hours in key characterization work.

Research uses

  • TREK-1 channel pharmacology
  • Rapid-onset antidepressant models (forced swim, novelty-suppressed feeding)
  • Hippocampal neurogenesis and BDNF upregulation
  • Serotonergic circuit research

Research dosing notes

Preclinical work uses microgram-range systemic dosing. Community research vials are typically 5–10 mg. No validated human dose exists.

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Half-life / kinetics

Short plasma; behavioral effects longer in rodents (~hours to a day)

Common research routes

Subcutaneous injection
Intranasal

Reported considerations

  • Unknown human adverse-effect profile
  • Theoretical cardiac TREK-1 expression concerns uncharacterized for this peptide
  • Injection or nasal irritation

Contraindications / cautions

  • Pregnancy
  • Active serious psychiatric illness requiring standard care
  • Unsupervised use as antidepressant substitute

Common research stacks

  • Semax
  • Selank
  • P21
  • DSIP

Legal / access note

Research chemical; not an approved antidepressant or nootropic drug.